Jason Gerberry -- Bank of America Merrill Lynch -- Analyst
Thanks, Elizabeth. Hey, guys. Maybe just for me, just on your selective TYK2. Can you talk a little bit about how you see the molecule differently than in Bristol's TYK2? And any important pharmacologic attributes of the molecule you think help differentiate and when we might get some more early stage data on that molecule. And then just on the situation with France and the female-only label, is that unique to France? I guess if something were surprising to happen that was discordant with the 13-week results. Just wondering if a female-only label is something that you view as a plausible label for the drug if something, again, discordant with the 13-week results were to pop up.
Walid Abi-Saab -- Chief Medical Officer and Member of Management Board
Okay. This is Walid. I'll take the two questions. So our molecule as a domain kinase inhibitor versus the messages in arsenic modulator. How will that translate in the clinic? Really is the big question, and for us, that's really where the money is at the end of the day. Preclinically, our molecule is selective and they're highly selective actually. And based on that, that's why we advanced it in development. In the clinical data and healthy subjects, we've had very good data from PK compatible with once a day dosing.
In addition, we've had some very good pharmacodynamic activity as well, which have confirmed what we have seen preclinically. And there's been no changes. As you know, we monitor these very carefully. We're quite similar with the JAK and mature in that space, changes in sort of lipid profile or changes in life of cells, so on and so forth. We have not seen anything with our compound again still within a Phase I healthy volunteer setting, but still, we can make healthy comparisons to our other molecules.
We are eagerly awaiting our Phase 1b study, which is, again, a small study, about 30 patients, two doses versus placebo in a 1-1-1 ratio. four weeks. This will give us a sense of how the compound is performing. But honestly, if you ask me what is the best way to compare it to Deucravacitinib is to run a rightsized Phase IIb trial similar to what they've done in psoriasis, most likely psoriatic arthritis but I think psoriasis would the one -- the area that's more validated or we have much more data, and then we can truly compare like-for-like both drug arms.
So as to sharing the data, we look forward to doing that at the earliest possibility and regarding mechanism of action, our target would be the upcoming hematology conference. I cannot promise 100% because I cannot promise that they will accept but that is our target. And the -- for the psoriasis data will be the first dermatology conference that will be coming up and we'd be talking at that.